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Morbus hansen tipe borderline lepromatous pada anak

Abstract

Leprosy or Morbus Hansen (MH) is a chronic infectious disease caused by the bacteria Mycobacterium leprae (M. leprae). Leprosy can occur at various ages from infants to old age with the most age groups, productive age. Prevalence and characteristics of leprosy in children are important in epidemiology and an indicator of the success of leprosy control. Disabilities obtained from an early age will affect the physical and mental development of child patient. The diagnosis of leprosy is based on four cardinal signs of leprosy, namely the presence of anesthesia, thickening of the nerve at the site of predilection, the presence of skin lesions, and the presence of acid fast bacilli (AFB) on the smear of the skin. A diagnosis of leprosy is made up of at least two of the first three cardinal signs or the fourth cardinal sign. In this case report reported cases of borderline lepromatous type leprosy in an 11-year-old boy. The diagnosis of leprosy is based on history, physical examination and investigations in the form of a acid fast bacilli (slit-skin smear). Therapy for the case include MDT MB children for 12 months.

Penyakit kusta atau Morbus Hansen (MH) merupakan penyakit infeksi kronis yang disebabkan oleh kuman Mycobacterium leprae (M. leprae). Penyakit kusta dapat menyerang berbagai usia dari bayi sampai usia lanjut dengan kelompok usia terbanyak adalah usia produktif. Prevalensi dan karakteristik penyakit kusta pada anak memiliki arti penting dalam epidemiologi dan merupakan indikator tingkat keberhasilan pengendalian penyakit kusta. Kecacatan yang didapat sejak usia dini akan mempengaruhi perkembangan fisik maupun mental penderita anak. Diagnosis penyakit kusta ditegakkan berdasarkan 4 tanda kardinal dari kusta yaitu adanya anestesia, penebalan saraf pada lokasi predileksi, adanya lesi kulit, dan didapatkan adanya Basil Tahan Asam (BTA) pada hapusan kulit. Diagnosis kusta ditegakkan minimal 2 dari 3 tanda kardinal pertama atau adanya tanda kardinal yang keempat. Pada laporan kasus ini dilaporkan kasus kusta tipe borderline lepromatous pada seorang anak lelaki berusia 11 tahun. Diagnosis kusta ditegakkan berdasarkan anamnesis, pemeriksaan fisik dan pemeriksaan penunjang berupa hapusan sayatan kulit (slit-skin smear). Terapi yang diberikan pada kasus meliputi MDT MB anak selama 12 bulan.

 

References

  1. Bryceson A, Platzgraff RE. Diagnosis. dalam: Leprosy. New
  2. York: Longman Group Limited; 1990: 57-75.
  3. Direktorat jenderal pengendalian penyakit dan penyehatan lingkungan. Epidemiologi. In: Buku Pedoman Nasional
  4. Pemberantasan Penyakit Kusta. Jakarta: Departemen
  5. Kesehatan RI; 2012.
  6. Anonim. Buku Register Kunjungan Sub Bagian Morbus
  7. Hansen, Poliklinik Kulit dan Kelamin Rumah Sakit Umum
  8. Pusat Sanglah. Denpasar; 2014-2016.
  9. WHO. Progress in leprosy control: Indonesia, 1991–2008.
  10. ; 85: 249–64. Di unduh dari http://www.who.int/wer.
  11. diakses December 10, 2017.
  12. Bryceson A, Platzgraff RE. Symtoms and signs. dalam:
  13. Leprosy. Edisi ke-3. New York: Longman Group Limited;
  14. : 25-56.
  15. Sehgal VN. Leprosy in children. dalam: Clinical Leprosy.
  16. New Delhi: Jaypee Brothers; 2004: 78-82.
  17. Sachdeva S, Amin SS, Khan Z, Alam S, Sharma PK.
  18. Childhood leprosy: a retrospective study. J Public Health
  19. Epidemiol. 2010; 2(9): 267-71.
  20. Rea TH, Modlin RL. Leprosy. In: Wolff K GL, Katz SI,
  21. Gilchrest BA, Paller AS, Leffell DJ, editors, ed. Fitzpatrick’s
  22. dermatology in general medicine. New York: McGraw Hill
  23. Companies; 2008: 1786-96.
  24. Imbiriba EB, Garnelo L, Levino A, Cunha MDG, Pedrosa V.
  25. Epidemiological profile of leprosy in children under 15 in
  26. Manaus (Northern Brazil), 1998-2005. Rev Saude Publica.
  27. ; 42(6): 1-5.
  28. Prashad P. Epidemiology. In: All about leprosy. New Delhi:
  29. Jaypee Brothers; 2005: 40-3.
  30. World Health Organization. WHO model prescribing information drugs used in leprosy. Geneva; 1998.
  31. Gupta R, Pandhi D. Case report: genital involvement and
  32. type I reaction in childhood leprosy. Lepr Rev. 2005; 76:
  33. -7.
  34. Sales AM, Leon APd, Duppre NC, Hacker MA, Nery JAC,
  35. Sarno EN, et al. Leprosy among patient contacts: a multilevel study of risk factors. PLoS Negl Trop Dis. 2011; 5(3):
  36. -6.
  37. Sahoo A, Singh PC, Pattnaik S, Singh N. Incidence of
  38. leprosy in school-children and their family members in
  39. Berhampur. Indian J Lepr. 2002; 74(2): 137-43.
  40. Prashad P. Classification. In: All about leprosy. New Delhi:
  41. Jaypee Brothers; 2005: 44-8.
  42. Weedon D. Bacterial and Rickettsial infections. dalam:
  43. Skin Pathology. UK: Elsevier; 2010: 547-72.
  44. Bryceson A, Platzgraff RE. Immunological complications:
  45. reactions. dalam: Leprosy. New York: Longman Group
  46. Limited; 1990: 115-26.
  47. Mochamad Hatta. European Journal: Epidemiology of leprosy on five isolated islands in the Flores Sea, Indonesia,
  48. September 2017.
  49. Direktorat jenderal pengendalian penyakit dan penyehatan lingkungan. Pencegahan dan tatalaksana cacat. In:
  50. Buku Pedoman Nasional Pemberantasan Penyakit Kusta.
  51. Jakarta: Departemen Kesehatan RI; 2006.h. 81-109.
  52. Amirudin MD, Hakim Z, Darwis E. Diagnosis Penyakit
  53. Kusta. Dalam Daili ES dkk editor. Kusta. Jakarta; Balai
  54. Penerbit FKUI; 2007.

How to Cite

Rahmawati, A., & Karmila, I. D. (2020). Morbus hansen tipe borderline lepromatous pada anak. Medicina, 51(2), 96–103. https://doi.org/10.15562/medicina.v51i2.812

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